PhD student in Molecular Cardiology.
Our laboratory is interested in molecular mechanisms of familial forms of heart failure, in particular in the pathogenesis of dilated cardiomyopathy (DCM) and the development of novel therapeutic strategies to combat heart failure.
The position will involve application of a state-of-the-art methodology (incl iPS technology, ChIP on chip, morpholino-based exon skipping technique etc) for identifying and antagonizing molecular mechanisms of heart failure in the human and mouse setting.
The position also includes a fully funded 3 months exchange programme with the Victor Chang Institute in Sydney, Australia. The successful candidates will have the opportunity to present their data on national and international conferences.
Your profile:
• University degree biology, molecular/cell biology, or a related field
• Experience in molecular and cellular techniques
• Highly motivated to learn new methodology, organized, and intellectually curious
• Fluency in English AND German
• Enjoys working in a young, multi-disciplinary team
The position is available immediately, supported by a grant of the Deutsche Forschungsgemeinschaft and is initially limited to 3 years.
Tübingen University, situated in the centre of Europe is a reputed and renowned address for international students. We offer a stimulating environment in a young research group. Please send a CV in German, copies of high school exam and university exams, and contact details of 2-3 referees along with a cover letter stating your reasons for applying to
Dr Michael Gramlich, email: michael.gramlich@med.uni-tuebingen.de.
Related publications:
Gerull B*, Gramlich M*, Atherton J et al. (2002). Mutation of TTN, encoding the giant filament titin, cause familial dilated cardiomyopathy. Nat Gen 30(2):201-204.
Gramlich M, Michely B, Krohne C, et al. (2009). Stress-induced dilated cardiomyopathy in a knock-in mouse model mimicking human titin-based disease. JMCC 47(3): 352-8.